Patients who undergo processes such as bone ...

Based on the causes of pneumonia, diseases are divided into the following types:

Bacterial pneumonia: As the name implies, this type of pneumonia caused by different types of bacteria. Most bacteria cause pneumonia is the pneumococcus. This type

pneumonia usually affects people who have weakened immune systems, for reasons as old age, disease, malnutrition, etc. Bacterial pneumonia indicated by signs like rapid breathing, increased pulse rate, high fever with chills and shivering, severe pain in the chest, knocking teeth, heart rate, greenish strattera cost color of rust and slime, heavy, etc. sweat of the most common types of pneumonia, atypical pneumonia. This type of pneumonia bacteria also induced. The bacteria responsible for causing SARS Chlamydophila pneumonia, Mycoplasma pneumonia and Legionella Pneumophila. Most forms of atypical pneumonia characterized by mild symptoms, but pneumonia caused by Legionella sometimes leads to severe symptoms. Mortality from pneumonia Legionella induced very high. Viral pneumonia: This option pneumonia can be caused by different viruses. The most common forms of viruses that cause viral pneumonia, influenza virus, parainfluenza virus, herpes simplex virus, rynovirusy, adenoviruses, Hantavirus, cytomegalovirus and respiratory syncytial virus. Symptoms of pneumonia are viruses induced as pneumonia episodes caused by bacteria. People with viral pneumonia are at risk of bacterial pneumonia. Pneumonia episodes caused by cytomegalovirus or CMV is more common among depressed immune individuals. CMV belongs to a group of herpes viruses. Patients who undergo processes like transplantation of bone marrow and solid organ transplantation are often infected with CMV. CMV pneumonia induced usually accompanied by any significant symptoms. However, if CMV causes pneumonia in AIDS patients, individuals undergoing chemotherapy, bone marrow or organ transplant patients, the consequences can be quite severe. Recently obtained statistics showed that 20 percent of patients undergoing bone marrow transplantation developed CMV pneumonia. Extremely high mortality rate among patients suffering from pneumonia, CMV simultaneously with hypoxemia (a condition characterized by low oxygen levels). Mycoplasma pneumonia: a condition called as it is caused by mycoplasma (tiny free-living agents of human diseases). Mycoplasmas are known to have signs as bacteria and viruses. People of all ages can suffer from mycoplasma pneumonia. The symptoms of mycoplasma pneumonia differ from symptoms

Further types of pneumonia. Patients may experience symptoms like rash and anemia. Some patients may develop neurological syndromes like encephalitis, myelitis, and meningitis. Aspiration pneumonia: This type of pneumonia occurs when people inhale foreign material. Inhaled foreign materials cause inflammation of the bronchi and lungs and can lead to the formation of pus in the lungs. Often inhaled foreign material content of the stomach, as the selection, vomiting, food and beverages. Problems with teeth, old age, with a sedative, as an alcoholic, coma, and anesthesia are among the factors of

aspiration pneumonia. The response of the lungs depends primarily on the number and nature of inhaled substances. The more acidic nature of the material, the greater the intensity of damage to the lungs. Fungal pneumonia: fungal pneumonia is one of the rarest types of pneumonia. Fungus causes this type of pneumonia pnevmotsistnoy. This type

pneumonia also more common in people with weakened immune systems. Often described as pnevmotsistnoy pneumonia complications faced by patients with AIDS. Patients with cancer are also at risk from pnevmotsistnoy pneumonia. Hospital pneumonia: Hospital pneumonia is a condition that occurs only at the expense of patient stay in hospital. Pneumonia: Often this condition is called CAP. CAP is classified as one of the worst forms of pneumonia. Pneumonia is the 4th most common cause of mortality in Britain and worth six in the same category in the United States. 85 percent of CAP episodes occur because of common pathogens as Haemophilus influenza, Moraxella catarrhal and pneumococcus. The remaining 15 percent of CAP cases occur due to atypical pathogens, as Legionella species, Mycoplasma pneumonia and Lobzyn. In rare cases the condition can be caused by Acinetobacter, Pseudomonas aeruginosa and Enterobacter. .

Sodium hydroxide is used to adjust the ph to 8.

Also >> << novel is a combination of a mixture of bile acids and salts indicator Ricinoleic acid, which can be used on Wednesday in the mechanism shown in addition to the automatic analyzer of microbes, in which the presence of Klebsiella and Enterobacter is snow environment. Detection broth of this invention contains from 2. From 3 to 2. 8% of the nutrients, about 0. 196 to about 0. 204% 3 - (alfa-acetonylbenzyl..)-4-hidroksikumarina and about 0. 049 to 0. 051% 2.4 dinitrofenil hydrazones at. alpha. -Ketohlutarovoyi acid that acts as a biological inhibitor suppresses the growth of E. coli, which usually gives good results in tests for Klebsiella and Enterobacter and are also working to stimulate growth of the organism Klebsiella. 2.4 dinitrofenil hydrazones at. alpha. -Ketohlutarovoyi acid also indicates positive growth of the organism Klebsiella. Nutritious part of Wednesday contains about 4. 5 to 5. 5 g / l Gelysate, approximately 9. 0 to 11. 0 g / l tsellobyoza, from about 9. 0 to 11. 0 g / l inositol, from about 0. 24 to 0. 26 g / L yeast extract, and from about 0. 24 to 0. 26 g / l D-biotin. Gelysate of BBL and gelatin hydrolyzate made by pancreatic digestion, and low in cystine and tryptophan. Suitable replacement Gelysate any source of nitrogen-free sugar. Inorganic nitrogen sources such as ammonium chloride, can also be used. Peptones contain small amounts of sugar can be used. Gelysate goal is to provide nitrogen in the body. Purpose tsellobyoza and inositol is to sugar in the body, which causes sugar fermentation sharp change of pH, which causes the indicator. An indicator of the present invention are sodium and dezoksyholatom Ricinoleic acid. The indicator works by creating a milky-white precipitate in an acidic environment. These changes are observed and recorded the mechanism described in Appendix automatic analyzer microorganisms. Indicator mixture also acts to suppress gram-negative microorganisms. From about 9. 0 to 11. 0 g / l sodium Ricinoleic acid. Sodium hydroxide is used to adjust the pH to 8. Is 0. The essence of the invention lies in the effects of chemical inhibitors, 3 - (alfa-acetonylbenzyl..)-4-hidroksikumarina and 2.4 dynytro phenyl hydrazones in. alpha. -Ketohlutarovoyi acid. These inhibitors act to inhibit the growth of microorganisms without Klebsiella. Growth of E. coli species, gram-positive bacteria and microorganisms that lead to high yield positives using traditional methods for detection of chemical inhibitors inhibited the process which uses an automatic analyzer microorganisms. The concentration of 3 - (. Alpha. Acetonylbenzyl-)-4-hidroksikumarina can be from about 0. 196 to about 0. 204%, and is most effective at 0. 2%. Concentration of 2.4 dynytro-phenyl hydrazones at. alpha. Ketohlutarovoyi-acid can be from about 0. 049 to 0. 051%, and is most effective at 0. 050%. If the concentration of any inhibitor is too low, a higher yield of unwanted false positives occurs. If the concentration is very high, profitability is lower. For preparation of 2. times. medium of 100 ml broth detection, Klebsiella and Enterobacter prepared by dissolving 0. 4 g of 3 - (. Alpha. Acetonylbenzyl-)-4-hidroksikumarina in 98 ml of distilled water mixed with 2 ml of 1 N sodium hydroxide solution. When 3 - (. Alpha. Acetonylbenzyl-)-4-hidroksikumarina dissolved, the following will be strattera dosing added to the solution:

Gelysate - 1. 0 g

tsellobyoza - 2. 0 g

Inositol - 2. 0 g

Yeast extract - 0. 05 grams

Sodium dezoksyholatom - 2. 0 g

D-biotin - 0. 05 grams

Sodium salt ritsinoleinovoy acid - 2. 0 g

2.4-dinitrofenil hydrazones at. alpha. Ketohlutarovoyi-acid - 5 mg

solution was stirred for 1:00. 2. 7 mg Green Diamond is added to a solution. PH brought to 8. 0 by using a drop of sodium hydroxide. Solution filter sterilized. Wednesday at double (2x) the usual concentration for use in wells and card described in the annex named automatic analyzer microorganisms. .

And by the way, talking about the basic ...

Superbakteriy is dastardly villains medical world: Theyre hard to catch and kill, and they nullify helpless. This week, reports CNN

appearance of resistant Klebsiella pneumonia karbapenemam (CRKP), error were considered rare on the west coast in Los Angeles. For seven months in 2010 were 356 cases of CRKP, in the county health department - mostly in health care facilities like hospitals and nursing homes. CNNs chief medical correspondent Elizabeth Cohen said that this is the first time CRKP studied in Los Angeles and the District of incidence was unexpectedly high, according to a press release the Company Health Care Epidemiology of America. It is not known how many people died from this bacteria, but >> << found that previous outbreaks caused 35% mortality. Although the error was officially registered in 36 states, health professionals find it all in other states too, where reporting is not required. Thus, it was only a matter of time when the agent in the news on the West Coast, according to Dr. Robert Moellering, and a specialist in infectious diseases at Harvard. We asked Moellering some basic facts about CRKP and resistance to antibiotics. Q: What is CRKPP? : CRKP means karbapenem resistant Klebsiella pneumonia, which simply means that the bacteria - gram-negative bacteria that type that is associated with common organisms found in the gastrointestinal tract, like E. coli

- Karbapenemam this resistance, the type of antibiotic, which until recently was the antibiotic of last resort. Sometimes it still is, but not always. Previously, karbapenemy will work against bacteria such as Klebsiella pneumonia >> << that were resistant to any other type of antibiotics. Bacteria develop resistance almost as fast as Weve managed to develop new antibiotics. Q: How CRKPs drug resistance work? CRKP strains contain enzymes that destroy karbapenemy. A huge problem, however, is a company that [CRKP] holds. These together with other bacteria that are multidrug-resistant, and they contain genetic elements, DNA fragments that can be transferred to those of other bacteria. [It]

Klebsiella pneumonia can transfer genetic material to resist and have done so in the case of E. coli

hospital-associated infections. Gram-negative [bacteria] are more difficult to treat with antibiotics, even without drug-resistant because they have more structural problems. For example, they have two outer membranes, as antibiotics, to go through two separate barriers to reach these effectively. They also contain several pump that pumped toxic substances such as antibiotics. Q: When there are first CRKP? : Ive known about these organisms for 10 years or so. They first appeared in North Carolina, caused great concern in New York in Manhattan. Theyd already spread to California, they are nothing new in California. There just seems to increase the number of infections as well as heard about it [now]. Q: How can we avoid CRKP infection? : Stay away from hospitals and nursing homes! No, you can not do it. This is not what

you have to do as what hospitals should do. First, they must identify these organisms, send cultures reputable laboratories. And then the next step is to follow the CDCs standard methods of isolation: quarantine infected people to take precautions, contact wear gowns and gloves and hand washing practices. And by the way, talking about big, very well, university affiliated hospitals [these problems]. And you probably go out and get on the street? Chances are low. But the reason [these mistakes] are so terrible, because they spread and mutate strattera 10mg, so that now many of them [including CRKP] susceptible only to [antibiotic] ​​kolistyn, which is very toxic and causes kidney damage. Some even get resistance to it, and then a few lines of defense. Thus, the mutation of bacteria in combination with a reduction in new drugs, you can see, we went to the collision. Q: Why Arent pharmaceutical companies allocation more resources on antibiotics? : Pharmaceutical companies largely withdrew from the business development of antibiotics. First of all, it is not easy, because all drugs have found simple. In addition, antibiotics actually treat patients - drug companies DONT always. Theyd, and develop statins and antidepressants, which you take for life, because they eventually sell more so Theres a big financial return for what you take for eight days, then never. In addition, there are large issues of regulation with the development of antibiotics in terms of obtaining FDA approval. They are taking steps to fix it, but they make it more difficult to do clinical research, they change the criteria for success at the crossing, they are not quite sure what their criteria. These are just some of the reasons why development has slowed, but very sad, because we desperately need new antibiotics. Q: Why be the epicenter of the hospital Superbug infections? : Hospitals contain patients and patients often seek infections with antibiotics. There is clearly some excessive use of antibiotics as well. You also have a group of patients who are more susceptible to infections, many of whom are immunocompromised, as they are undergoing chemotherapy or organ transplants - things that change their ability to fight infection. Another important factor displacement. Even in the best circumstances, some types of infection are required to pass. So that all hospitals can do right now - but just close your doors - it is best to try to follow the principles of infection control, as described in the CDC. If you do this and do it very well, the number of infected people will go down. I can not imagine there is not the main hospital, not trying to do this is though - it's not easy. Related links: CNNs Commissioner patient >> <<

In the form of microbial sex called transformation ...

Bacteria acquire resistance genes in three directions. A. A spontaneous mutation, DNA bacteria can change spontaneously, as indicated in star formation. Drug-resistant tuberculosis arises this way. 2. In the form of microbial sex called transformation, one bacterium can take up DNA from another. Gonorrhea resistant to penicillin (

) the results change. 3. Most frightening, however, the resistance acquired in a small circle of DNA called plasmids. Plasmids can buzz among bacteria of different species - they do not apply - and to some resistance. In 1968, 12,500 died in hvatemaltsiv epidemic diarrhea Shigella, caused by the parasite harboring plasmids that conferred resistance to four antibiotics! But bacteria do something much more clever than just mutated. This is risky because bacteria prefer to share the secrets of biochemical - resistance genes - that will enable them to resist or destroy antibiotics. This diabolical barter can occur in several ways. A. Plasmids - small fragments of DNA as a mini-chromosome - that exist outside the main chromosome. This exchange can jump wide units in bacterial phylogeny (

). It's almost as if a cow could provide genes crow and taught it growing teeth. 2. Gene cassettes

genes that may be linked to chromosomes (). Although the mechanism of this complex is comparable with a trip to the mall, says Davis. Genes called integrones code for enzymes called integrase, which can splice these cassettes in the chromosomes or other genetic material, where they become functional. It does something integrones features like shopping cart, says Davis. If bacteria can only resist, taking gene cassettes, then, as shoppers in the store video, not sure what they saw their first choice, the bacteria can pick up a few tapes and get some resistance to antibiotics. Besides, says Abigail Salyers microbiologist at the University of Illinois, "Bacteria also integrate resistance of disinfectants and contaminants in these clusters. Thus, the use of disinfectants or pollution can select for resistance to antibiotics that may be exactly what

found back in the beginning of our history. From the human point of view, the problem with this kind of resistance to its sustainability. Once lodged in chromosomes or plasmids, these resistance genes are distributed normal genes are daughters (

) cells. "The Origin and interstate spread of ... "In >> << gives a detailed picture of how the deadly bug that causes tuberculosis acquired resistance to many antibiotics. So far we have talked in general about resistance without painting a good idea of ​​how it works. And here we are in for another surprise, another sign that could be called a microbial mind. It appears that resistance to antibiotics is part of a broader picture of the way bacteria protect themselves against chemical threats in their environment.

It seems that these protective mechanisms is surprisingly similar to many kinds of organisms -. many threats "Question resistance converges with human infectious diseases and agricultural angles," says Joe Phytopathology Handelsman "or microbe is trying to protect themselves from antibiotics, fungicides, insecticides, herbicides, even of antiviral drugs. ". Handelsman, who studies the interaction between fungi and bacteria at the University of Wisconsin-Madison, shows more similarity "At the molecular level, there are few mechanisms of resistance: Change the target molecule, inactivation or spread of drugs or pesticides, sequester (

) or drug pesticides, or keep it in a cage "from the beginning. Overall, it makes sense that the microbes would defense. Finally, in the course of billions of years on the planet, they overcome the countless dangers of chemicals. But what is the source of resistance genes to antibiotics in the first place? Perhaps the organisms that initially produced antibiotics. While it may seem strange, it logically. Suppose that I, lowly bacteria to make a chemical that, say, destroys the bacterial cell wall. Do I need some protection against chemical that children are sometimes called "a drug? "

This assumption is not only logical, it may even be true, says Davis." We believe that resistance genes in actinomycetes (bacteria that produce many antibiotics) that have the same biochemical function of resistance genes "in the samples from hospital patients with genetic sequences similar -. but not identical - it suggests that genes jump between species, although Davies admits that "we can not prove it. "

Salyers, who studies the process of genes between species jumps, says that bacteria have a lot of tricks to move the resistance genes on the one hand, they seem to cause other bacteria to start the genetic swap meet. When plasmid DNA resistance released bacteria taken other bacteria, the receiver can be stimulated to release their plasmids, a process called retrotransfer ". This enables the transmission of bacteria, the ability to sample the DNA of other bacteria, "says Salyers. She said the relationship is a new form of symbiosis (

). As if this prospect bacteria merge win antibiotics not alarming enough to recognize that this generosity extends beyond the members of their own species, Salyers said. " Virtually all bacteria can get genes from any other bacteria. "

What is the evidence of this movement? Scientists are distantly related bacteria with resistance genes, DNA (

) is from 95 to 99 percent identical. Although it is highly unlikely by chance alone, a strong suggestion - but not proof -. That genes have a buy strattera common origin, but do not forget that anti-bacterial antibiotic toolkit also includes several mutations that could explain that happened in New York, where >> << deadly drug resistant was spit.

Gonococcal and chlamydial conjunctivitis ...

Conjunctivitis: bacterial, viral,

to consider all forms of conjunctivitis including bacterial, viral, allergic and other types of inflammation include transparent, mucous membranes (

) in the white part or . Infectious causes inflammation of the eyes and conjunctivitis include bacteria, viruses and fungi. Noninfectious causes include allergies, foreign bodies and substances. The phrase "pink eye" is usually used for conjunctivitis, because liberalism or redness of the conjunctiva is one of the most noticeable symptoms. Bacterial conjunctivitis is a common type of pink eye caused by bacteria that infect the eye through various sources of pollution. Bacteria can be transmitted through contact with an infected person, the impact of contaminated surfaces or by other means, such as sinus or ear infections. The most common types of bacteria that cause bacterial conjunctivitis include Staphylococcus aureus, Haemophilus influenzae, Streptococcus pneumoniae and Pseudomonas aeruginosa. Bacterial conjunctivitis usually produces a thick discharge or pus and may affect one or both eyes. As with any bacterial infection, antibiotics are necessary to eliminate the bacteria. Treatment of bacterial conjunctivitis is usually carried out with topical antibiotics >> << and / or eye ointments. Treatment usually takes from one to two weeks, depending on the severity of infection. Viral conjunctivitis is another common type of pink eye that is because viruses can spread air through sneezing and coughing. Viral conjunctivitis may also accompany common viral upper respiratory tract infections such as measles, flu or cold. Viral conjunctivitis usually produces a watery discharge. Usually the infection starts in one eye and rapidly spreads to the other eye. Unlike bacterial infections, antibiotics will not work against viruses. No eye drops or ointment effective against common viruses that cause viral conjunctivitis. But viral conjunctivitis involuntarily, and thus disappears by itself after some time. Typically, with viral conjunctivitis, third to fifth days of the worst. Then his eyes begin to improve themselves. Treatment of viral conjunctivitis usually includes support for treatments such as eye drops to help reduce symptoms such as vasoconstrictor for whitening eyes, decongestants to reduce swelling of the surface and antihistamines to reduce the itching. Treatment usually lasts one to two weeks, depending on the severity of infection. Gonococcal and chlamydial conjunctivitis is bacterial forms associated with infections, sexually transmitted diseases, including gonorrhea and chlamydia. Newborn children can be exposed when they pass through the birth canal infected mother. Trachoma is a form of chlamydial infection, which causes scarring on the surface of the eye. Trachoma is the leading cause of world blindness prevented. Conjunctivitis of the newborn found in infants can lead to blindness if not treated. Up to 10 percent of all pregnant women in the United States are sexually transmitted chlamydial infections. If the infection is not treated mothers, the probability that a newborn will develop related ocular infection ranges from 10 to 20 percent. *

Another type of sexually transmitted diseases associated with herpes simplex virus type 2, is located in the genital area can infect the eyes of children, as they are born. Herpes simplex virus type 1 cause herpes in the mouth and can lead to type >> << that causes pink eye. If you are pregnant and suspect you may have a sexually transmitted disease, you should be checked and possibly treat any infection before birth. In the United States, antibiotic ointment is often used as the primary standard for the care of newborns to prevent exactly. Allergic conjunctivitis is caused by very often. Eye allergies, like other species may be caused by allergens including pollen, pet dander and dust mites. The most common symptom of allergic conjunctivitis have itchy eyes, which can be removed with special eye drops containing antihistamines to control the allergic reaction. These eye drops are available without a prescription and a doctor's prescription. Avoid allergens are also important in the treatment of allergic conjunctivitis. Allergic conjunctivitis may be seasonal or perennial (all year), depending on the allergen causing the reaction. Giant papillary conjunctivitis (GPC) usually involves both eyes and often affects the soft contact lens owners. This condition can cause contact lens intolerance, itching, heavy discharge, tearing and red spots on the inside of the eyelids. You need to stop wearing your, at least for a while. Your

may also recommend you go to a different type of contact lenses to reduce the likelihood of conjunctivitis coming back. For example, you have to strattera 40mg switch between >> << to this or vice versa. Or you may have to try the type of lens that you replace frequently, for example. CPC may also be the result of an artificial eye (prosthesis), stitches and more. Your eye doctor determines removal is necessary. Noninfectious conjunctivitis causes of eye irritation that can result from many sources, including smoke, exhaust gases of diesel engines, spirits and some chemicals. Some forms of conjunctivitis as a result of sensitivity to certain substances ingested, including herbs such as turmeric and ochanky. **

Some forms of pink eye, including giant papillary conjunctivitis may be caused by immune reactions of the eyes, such as a reaction to wearing contact lenses or eye prosthesis (artificial eye). The reaction to the preservatives in eye drops or ointment may also cause toxic conjunctivitis. and the first to know about eye care news and new articles. * Principles and Practice of Infectious Diseases, 6th ed. (2005)

** American Journal of Ophthalmology, October 2004.

Gram-negative bacteria can be stubborn infections ...

Gram-negative bacteria are bacteria that are not converted into violet Gram staining process is the main step in identifying bacteria. Most bacteria are divided into either gram positive or gram-negative bacteria that express the basic differences in the composition of their cell wall. These differences often have a direct impact on what makes bacteria, some Gram-negative bacteria, pathogens in nature. Was developed in 1884 by Hans Christian Gram. In this process bacteria fixed on a slide, and then bathed in crystal violet, basic dye solution. All cells on a slide strattera turn purple, then hydrate like

added to fix the color. Then decolorizer added to the slide. If gram-negative bacteria, decolorizer shut crystal violet, TK permeable cell walls do not allow crystal violet staining of bacteria. Then, secondary spot, turning gram-negative bacteria, pale pink, but has no impact on the already purple gram-positive bacteria. These bacteria have thin cell walls with the outer layer composed of proteins and lypopolysaccharide. This outer layer sometimes reacts, causing

and infection. In addition to preventing bacterial outer membrane staining cells and helps the bacteria to resist the assortment of drugs, making treatment of infections with Gram-negative bacteria, and difficult. Some examples of gram-negative bacteria include, and. Many other >> << and gram-negative, including some forms of bacterial sources of gastrointestinal disorders, and spirochetes. Gram-negative bacteria can be stubborn infectious agents, and many sources of deadly infections are gram-negative, including the bacteria that contribute to secondary infection in hospitals and clinics. Gram staining can give an insight into the cell wall labels, so this is a normal step in exploring new bacteria in the laboratory. Once the bacteria were exposed to Gram, additional studies are needed to determine the bacteria source, and as infection caused by bacteria can be seen, but Gram makes a good first step. Spot has the added advantage of highlighting key structures of bacteria, including the internal structure of cells, making them easier to see and understand. Gram staining does not work for all bacteria, but Gram-and Gram-negative bacteria uncertain variables can not be defined as follows. .

This type of treatment is different from ...

When it comes to bacterial vaginosis, usually there are two approaches to treatment: There are the usual >> << bacterial vaginosis treatment, then we have alternative treatment options, too. If you visit the doctor very likely that it will follow, and with traditional methods of treatment which treat BV with antibiotics. Antibiotics are then taken orally, some forms can also be used externally. In addition, antibiotics can sometimes be quite expensive as well. But this is not a problem with antibiotics. The strattera no prescritpion problem is that antibiotics, but in fact brute force treatment. We know that antibiotics are effective in killing bacteria in the vagina, there is no doubt. But antibiotics do not take into account the reasonable and very important balance of microorganisms in the vagina. This type of treatment did not differ from the good bacteria and bad bacteria. Just the elimination of vaginal bacteria, they generally ignore the importance of restoring and maintaining healthy, natural environment of the vagina. Violation otherwise balanced and healthy environment microorganisms cause outbreaks of bacterial vaginosis in the first place. Typically, women's body may adjust and regulate itself in this regard is the natural mechanism that keeps infections like bacterial vaginosis did not appear. Only in cases where a natural balance is off, we see infections. The reason for this violation and entering BV may be overdoing it with fragrant body washes, showers, sinks and tubs that contain certain chemicals, but a few examples. Cure Your BV in 3 days! Or your money back, guaranteed! Click to try it 100% risk free! .